Does Symettrel induce euphoria?
as it is a dopamine agonist :D
(dopmaine is the PLEASURE chemical in the brain)
Answers:
No, I don't think it is active within the mesolimbic reward pathway.
There is no Symettrel. Symmetrel (contains AMANTADINE) treats and prevents certain types of flu. It is also treats Parkinson disease and uncontrolled muscle movements caused by some medicines
Amandatine is adjectives in the treatment of Parkinson's syndrome and in the short-term management of drug-induced extrapyramidal symptoms.
In Parkinson's syndrome, amantadine have been used alone and in combination with anticholinergic antiparkinson drugs and beside levodopa. The final therapeutic benefit seen with amantadine is significantly smaller amount than that seen with levodopa. The maximal therapeutic benefit to be obtain with amantadine is usually seen within 1 week. However, initial benefits may diminish next to continued dosing.
Amantadine is useful as an adjunct in patients who do not tolerate optimal doses of levodopa alone or contained by combined therapy with a decarboxylase inhibitor. In these patients, the addition of amantadine may result within better control of Parkinson's syndrome and may help to smooth out fluctuations in performance.
The comparative efficacy of amantadine and anticholinergic antiparkinson drugs have not yet been established. When amantadine or anticholinergic antiparkinson drugs are each used near marginal benefit, concomitant use may permit the same degree of control, normally with a lower dose of the anticholinergic medication.
Amantadine is effective in reducing severity or abolish drug-induced extrapyramidal reactions including parkinsonism syndrome, dystonia and akathisia. It is not effective in the regulation of tardive dyskinesia.
Although anticholinergic-type side effects have been noted when used in patients beside drug-induced extrapyramidal reactions, there appears to be a lower incidence of these side effects than that observed with anticholinergic antiparkinson drugs.
Antiparkinsonian agents should not usually be used prophylactically during neuroleptic control. However, they may be given when needed to suppress extrapyramidal symptoms. Therefore, amantadine may be used in the management of extrapyramidal symptoms which cannot be controlled by reduction of neuroleptic dosage, but should be discontinued as soon as it is no longer required. Amantadine should be withdrawn after a length of time to determine whether there is recrudescence of extrapyramidal symptoms. Source(s): http://www.mentalhealth.com/drug/p30-s05…
alex
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(dopmaine is the PLEASURE chemical in the brain)
Answers:
No, I don't think it is active within the mesolimbic reward pathway.
There is no Symettrel. Symmetrel (contains AMANTADINE) treats and prevents certain types of flu. It is also treats Parkinson disease and uncontrolled muscle movements caused by some medicines
Amandatine is adjectives in the treatment of Parkinson's syndrome and in the short-term management of drug-induced extrapyramidal symptoms.
In Parkinson's syndrome, amantadine have been used alone and in combination with anticholinergic antiparkinson drugs and beside levodopa. The final therapeutic benefit seen with amantadine is significantly smaller amount than that seen with levodopa. The maximal therapeutic benefit to be obtain with amantadine is usually seen within 1 week. However, initial benefits may diminish next to continued dosing.
Amantadine is useful as an adjunct in patients who do not tolerate optimal doses of levodopa alone or contained by combined therapy with a decarboxylase inhibitor. In these patients, the addition of amantadine may result within better control of Parkinson's syndrome and may help to smooth out fluctuations in performance.
The comparative efficacy of amantadine and anticholinergic antiparkinson drugs have not yet been established. When amantadine or anticholinergic antiparkinson drugs are each used near marginal benefit, concomitant use may permit the same degree of control, normally with a lower dose of the anticholinergic medication.
Amantadine is effective in reducing severity or abolish drug-induced extrapyramidal reactions including parkinsonism syndrome, dystonia and akathisia. It is not effective in the regulation of tardive dyskinesia.
Although anticholinergic-type side effects have been noted when used in patients beside drug-induced extrapyramidal reactions, there appears to be a lower incidence of these side effects than that observed with anticholinergic antiparkinson drugs.
Antiparkinsonian agents should not usually be used prophylactically during neuroleptic control. However, they may be given when needed to suppress extrapyramidal symptoms. Therefore, amantadine may be used in the management of extrapyramidal symptoms which cannot be controlled by reduction of neuroleptic dosage, but should be discontinued as soon as it is no longer required. Amantadine should be withdrawn after a length of time to determine whether there is recrudescence of extrapyramidal symptoms. Source(s): http://www.mentalhealth.com/drug/p30-s05…
alex
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