How does an increase contained by intracranial pressure decline respiratory rate?
I just read this (and my book doesn't explain of course), and I cannot put the two together. I understand that intracranial pressure is a result of CSF, brain tissue, and blood , but I cannot relate this to the respiratory system. Use any chemistry/physiology/ or whatever explanation you can to abet me better understand this. Detailed answers are extremely welcome.
Answers:
Increased intracranial pressure increased pressure to your respiratory center which may slow down your respiration.
Atstdriver have it exactly right.
I see lots of neurosurgical patients with elevated ICP however they are usually intubated by the time they hit the ICU so respiratory issues are already taken care of. Source(s): PA surgical critical care
It's due to direct pressure on the brainstem; as intracranial pressure increases, you start to get herniation, and as a result, the brainstem (where the respiratory centers are) get compressed and you begin to have respiratory dysfunction (it actually could be any faster or slower, but the point is that it is irregular). The respiratory changes are actually the third of a group of signs known as Cushing's triad, the other two of which are hypertension and bradycardia (increased BP from sympathetic stimulation due to hypoperfusion of the brain, and bradycardia due to the baroreceptor reflex). When you see those three together, it's pretty indicative of greatly increased ICP and that things are roughly to turn really bad, really fast for that patient.
Cocaine is a hell of a drug
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Answers:
Increased intracranial pressure increased pressure to your respiratory center which may slow down your respiration.
Atstdriver have it exactly right.
I see lots of neurosurgical patients with elevated ICP however they are usually intubated by the time they hit the ICU so respiratory issues are already taken care of. Source(s): PA surgical critical care
It's due to direct pressure on the brainstem; as intracranial pressure increases, you start to get herniation, and as a result, the brainstem (where the respiratory centers are) get compressed and you begin to have respiratory dysfunction (it actually could be any faster or slower, but the point is that it is irregular). The respiratory changes are actually the third of a group of signs known as Cushing's triad, the other two of which are hypertension and bradycardia (increased BP from sympathetic stimulation due to hypoperfusion of the brain, and bradycardia due to the baroreceptor reflex). When you see those three together, it's pretty indicative of greatly increased ICP and that things are roughly to turn really bad, really fast for that patient.
Cocaine is a hell of a drug
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